CHAPEL HILL -- New research at the University of North Carolina at Chapel Hill has found that a fatty molecule on the surface of platelet cells is a regulator of blood coagulation. Besides its role in forming cell membranes, the lipid phosphatidylserine, or PS, also appears to activate the final step in the process of blood coagulation - the conversion of prothrombin to thrombin, the central enzyme of coagulation. Thrombin is needed both to encourage and then cut off clotting at a wound.
The above story is based on materials provided by University Of North Carolina School Of Medicine. Note: Materials may be edited for content and length.
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