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Fat Cell Defect May Trigger Insulin Resistance In Muscle And Liver

Date:
February 8, 2001
Source:
Beth Israel Deaconess Medical Center
Summary:
Fat cells that can't take up blood sugar normally appear to trigger the same problem in muscle and a related problem in liver, say researchers from Beth Israel Deaconess Medical Center in Boston. Using a sophisticated genetic approach, the researchers wiped out the key protein in fat cells that insulin tells to move glucose from the blood into the cells. Soon, muscle also ignored insulin's orders to take up glucose, and liver disregarded insulin's instructions to shut down the organ's glucose production.

Boston -- Fat cells that can't take up blood sugar normally appear to trigger the same problem in muscle and a related problem in liver, say researchers from Beth Israel Deaconess Medical Center in Boston. Using a sophisticated genetic approach, the researchers wiped out the key protein in fat cells that insulin tells to move glucose from the blood into the cells. Soon, muscle also ignored insulin's orders to take up glucose, and liver disregarded insulin's instructions to shut down the organ's glucose production.


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The above story is based on materials provided by Beth Israel Deaconess Medical Center. Note: Materials may be edited for content and length.


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Beth Israel Deaconess Medical Center. "Fat Cell Defect May Trigger Insulin Resistance In Muscle And Liver." ScienceDaily. ScienceDaily, 8 February 2001. <www.sciencedaily.com/releases/2001/02/010208074012.htm>.
Beth Israel Deaconess Medical Center. (2001, February 8). Fat Cell Defect May Trigger Insulin Resistance In Muscle And Liver. ScienceDaily. Retrieved April 19, 2014 from www.sciencedaily.com/releases/2001/02/010208074012.htm
Beth Israel Deaconess Medical Center. "Fat Cell Defect May Trigger Insulin Resistance In Muscle And Liver." ScienceDaily. www.sciencedaily.com/releases/2001/02/010208074012.htm (accessed April 19, 2014).

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