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Duke Researchers Identify The Mechanisms By Which Drug Combinations Kill Fungal Infections

Date:
February 12, 2002
Source:
Duke University Medical Center
Summary:
Molecular biologists in the Mycology Research Unit at Duke University Medical Center have traced cellular pathways that are targeted to enhance the action of drugs used to treat fungal infections in people with compromised immune systems, such as those undergoing organ and bone marrow transplants. The discovery of how the immune-suppressing drugs enhance the action of antifungal drugs could lead to the development of new drugs designed to treat fungal infections such as systemic yeast infections and often-deadly cryptococcal infections.

DURHAM, N.C. – Molecular biologists in the Mycology Research Unit at Duke University Medical Center have traced cellular pathways that are targeted to enhance the action of drugs used to treat fungal infections in people with compromised immune systems, such as those undergoing organ and bone marrow transplants. The discovery of how the immune-suppressing drugs enhance the action of antifungal drugs could lead to the development of new drugs designed to treat fungal infections such as systemic yeast infections and often-deadly cryptococcal infections.


Story Source:

The above story is based on materials provided by Duke University Medical Center. Note: Materials may be edited for content and length.


Cite This Page:

Duke University Medical Center. "Duke Researchers Identify The Mechanisms By Which Drug Combinations Kill Fungal Infections." ScienceDaily. ScienceDaily, 12 February 2002. <www.sciencedaily.com/releases/2002/02/020207075321.htm>.
Duke University Medical Center. (2002, February 12). Duke Researchers Identify The Mechanisms By Which Drug Combinations Kill Fungal Infections. ScienceDaily. Retrieved April 24, 2014 from www.sciencedaily.com/releases/2002/02/020207075321.htm
Duke University Medical Center. "Duke Researchers Identify The Mechanisms By Which Drug Combinations Kill Fungal Infections." ScienceDaily. www.sciencedaily.com/releases/2002/02/020207075321.htm (accessed April 24, 2014).

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